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Glycinate Propionyl-L-Carnitine HCl ( ) is a form of propionyl-L-carnitine bonding to the amino acid glycine at the molecular level. Propionyl L-Carnitine is a derivative of the amino acid L-Carnitine used to metabolize and transport stored body fat for use as energy. The addition of glycine molecules increases the bioavailability of propionyl L-Carnitine and can also act as a potent nitric oxide accelerator, resulting in a wide range of benefits.
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GPLC as an effective form of L-Carnitine (ordinary L-Carnitine is an ineffective form, over-metabolizing of L-Carnitine by microorganisms in the small intestine, )
GPLC helps the body produce energy from stored body fat by transporting fatty acids from fat cells into the mitochondria of muscle cells, where fatty acids can be metabolized and used to produce energy. This process is called fat metabolism. Increasing the use of stored body fat for energy will result in a decrease in stored body fat.
GPLC can also increase levels of nitric oxide in the blood, resulting in increased blood flow and increased oxygen and nutrients delivered to working muscles. Therefore, when supplementing with GPLC, users may experience increased vasodilation and muscle pumping sensation.
Combined with a sensible diet and exercise regimen, GLPC can help improve fat metabolism, increase blood flow and muscle pumping, improve strength, and promote recovery.
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To take full advantage of GPLC, it is best to take it 30-45 minutes before exercise. It can be taken alone or with any other pre-workout supplement.
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GPLC begins working immediately after entering the bloodstream, usually 15-30 minutes after consumption. Users can expect to experience an increase in muscle pumps and an increase in vascular distribution during a workout.
bibliography:
1. Speth, John D & Spielmann, Katherine. (2001). Energy Source, Protein Metabolism, and Hunter-Gatherer Subsistence Strategies. Journal of Anthropological Archaeology. 2. 1-31.
2. Olsen N.(2018), What Is Protein Poisoning?, The request could not be satisfied, Last viewed: November 20, 2020[4] Noli D, Avery G (2004) Protein poisoning and coastal subsistence. Journal of Archaeological Science 15: 395–401
3. Maersk M., Belza A., Stødkilde-Jorgensen H., Ringgaard S., Chabanova E., Thomsen H., Pedersen S. B., Astrup A., and Richelsen B. (2012) Sucrose-sweetened beverages increase fat storage in the liver, muscle, and visceral fat depot: a 6-mo randomized intervention study. Am. J. Clin. Nutr. 95, 283–289
4. Ackerman Z., Oron-Herman M., Grozovski M., Rosenthal T., Pappo O., Link G., and Sela B. A. (2005) Fructose-induced fatty liver disease: hepatic effects of blood pressure and plasma triglyceride reduction. Hypertension 45,1012–1018 10.1161/01.HYP.
5. Ishimoto T., Lanaspa M. A., Le M. T., Garcia G. E., Diggle C. P., Maclean P. S., Jackman M. R., Asipu A., Roncal-Jimenez C. A., Kosugi T., Rivard C. J., Maruyama S., Rodriguez-Iturbe B., Sánchez-Lozada L. G., Bonthron D.T., et al. (2012) Opposing effects of fructokinase C and A isoforms on fructose-induced metabolic syndrome in mice. Proc. Natl. Acad. Sci. U.S.A. 109, 4320–4325.